腮腺炎病毒IgG ELISA診斷試劑盒(美國FOCUS)

廣州健侖生物科技有限公司

（廣州健侖生物科技有限公司是集研制開發、銷售、服務于一體的高新技術企業,公司產品涉及臨床快速診斷試劑、食品安全檢測試劑，違禁品快速檢測，動物疾病防疫檢測試劑，免疫診斷試劑、臨床血液學和體液學檢驗試劑、微生物檢驗試劑、分子生物學檢驗試劑、臨床生化試劑、有機試劑等眾多領域，同時核心代理Panbio、FOCUS、Qiagen、IBL、CORTEZ、Fuller、Inbios、BinaxNOW、LumuQuick、日本富士、日本生研等多家著名診斷產品集團公司產品，致力于為商檢單位、疾病預防控制中心、海關出入境檢疫局、衛生防疫單位,緝毒系統，戒毒中心，檢驗檢疫單位、生化企業、科研院所、醫療機構等機構與行業提供*、高品質的產品服務。此外，本公司還開展食品、衛生、環境、藥品等多方面的第三方檢測服務。）

廣州健侖長期供應各種PCR試劑盒，主要代理進口和國產品牌的流行病毒PCR檢測試劑盒。例如：甲乙型流感病毒核酸檢測試劑盒、黃熱病毒核酸檢測試劑盒、諾如病毒核酸檢測試劑盒、登革病毒核酸檢測試劑盒、基孔肯雅病毒核酸檢測試劑盒、結核桿菌核酸病毒檢測試劑盒、孢疹病毒核算檢測試劑盒、西尼羅河病毒PCR檢測試劑盒、呼吸道合胞病毒核酸檢測試劑盒、冠狀病毒PCR檢測試劑盒等等。蟲媒體染病系列、呼吸道病原體系列、發熱伴出疹系列、消化道及食源感染系列。

產品規格：96T/盒

存儲條件：4-8℃

我司同時還提供、美國FOCUS、西班牙DIA、美國trinity等試劑盒：

麻疹、風疹、甲流 、乙流、單皰疹1型、單皰疹2型、百日咳、百日咳毒素、腮腺炎、帶狀皰疹、單純皰疹、HSV1型特異性、巨細胞-特異、風疹-特異、弓形蟲-特異、棘球屬、嗜肺軍團菌、破傷風、蜱傳腦炎、幽門螺旋桿菌、白色念珠菌、博氏疏螺旋體、細小病毒、鉤端螺旋體、腺病毒、Q熱柯克斯體、煙曲霉菌、埃可病毒、EB病毒、衣原體、耶爾森菌、空腸彎曲桿菌、炭疽桿菌、白喉、腸道病毒、柯薩奇病毒、肺炎衣原體、沙眼衣原體、土拉弗朗西斯菌、漢坦病毒、類風濕因子、呼吸道合胞病毒、單純皰疹病毒質控品、巨細胞質控品、弓形蟲質控品、風疹麻疹質控品、等試劑盒以。

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腮腺炎病毒IgG ELISA診斷試劑盒(美國FOCUS)

我司還提供其它進口或國產試劑盒：登革熱、瘧疾、流感、A鏈球菌、合胞病毒、腮病毒、乙腦、寨卡、黃熱病、基孔肯雅熱、克錐蟲病、違禁品濫用、肺炎球菌、軍團菌、化妝品檢測、食品安全檢測等試劑盒以及日本生研細菌分型診斷血清、德國SiFin診斷血清、丹麥SSI診斷血清等產品。

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腮腺炎病毒IgG ELISA診斷試劑盒(美國FOCUS)

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【公司名稱】 廣州健侖生物科技有限公司
【市場部】     歐

【】 
【騰訊  】 
【公司地址】 廣州清華科技園創新基地番禺石樓鎮創啟路63號二期2幢101-103室

急性呼吸窘迫綜合征
是由心源性以外的各種肺內外致病因素導致的急性、進行性缺氧性呼吸衰竭，臨床表現為頑固性低氧血癥、呼吸頻率加快和呼吸窘迫，X線胸片顯示雙肺彌漫性浸潤影,后期多并發多器官功能衰竭，血氣分析示氧合指數(指動脈血二氧化碳分壓/用力吸氧量)等于或小于2617 kPa。

急性左心衰竭引起的嚴重肺淤血和水腫
有心臟基礎疾病，有左心衰竭的癥狀、體征，胸片提示肺淤血和水腫，有時和肺炎、慢性阻塞性肺疾病(慢阻肺)很難鑒別，但左心衰竭的病人血中腦鈉肽水平升高，而后兩者腦鈉肽水平不高，有助于鑒別。

彌漫性肺間質纖維化
臨床表現為進行性呼吸困難伴有刺激性干咳，X線胸片雙中下肺彌漫性網狀細菌影，肺功能檢查為限制性肺通氣功能障礙。

胸膜疾病
包括大量胸腔積液、氣胸，胸膜增厚。此類疾病突出的臨床表現是與體位無關的呼吸困難，體格檢查示氣管可移位，胸腔受累部位肋間隙可增寬或變窄，叩診呈濁音或鼓音，肺功能檢查為限制性肺通氣功能障礙，胸片、肺CT檢查有胸腔積液、積氣或胸膜增厚的表現。

急性肺栓塞
是由于內源性或外源性栓子堵塞肺動脈或其分支引起肺循環障礙的臨床綜合征。有兩個基本臨床癥候群：肺梗死:突發呼吸困難、胸痛、咯血、胸膜摩擦音，不能解釋的呼吸困難。該病有引起栓子形成或高凝狀態的基礎疾病，胸片顯示肺出現卵圓形或三角形浸潤細菌影，重癥者肺動脈段突出，實驗室檢查可見血D-二聚體水平升高而肌鈣蛋白水平不高，心電圖可有異常，心、肺動脈造影及放射性核素肺灌注掃描可準確了解栓塞所在的部位及范圍。

周圍循環缺血性疾病常見于各種原因引起的嚴重休克，由于周圍血管收縮心排血量減少，循環血容量不足，或周圍動脈縮窄，周圍組織血流灌注不足，缺氧致皮膚黏膜呈青紫色。新生兒嚴重腹瀉，如果治療不當，由于脫水，亦可造成紫紺。再者，雷諾病、肢端發紺癥亦屬此類。雷諾病的特點是雙手或雙足有麻木或厥冷感，發作時各指(趾)出現典型的皮膚蒼白-青紫-潮紅的雷諾現象。手足紫紺癥的特點主要是在常溫下持續的毛細血管前小動脈痙攣，導致血流減少，皮膚青紫和皮溫降低，紅外熱成像熒屏為暗黑色細菌影或不顯影。

周圍循環淤血性疾病
如右側心力衰竭、縮窄性心包炎以及局部靜脈病變(血栓性靜脈炎、上腔靜脈綜合征、下肢靜脈曲張)，其發生紫紺的機制是體循環淤血，周圍血流緩慢，氧在周圍組織被攝取過多所致。

Acute respiratory distress syndrome
Is caused by acute and progressive hypoxic respiratory failure caused by a variety of factors outside the cardiogenic lung, clinical manifestations of refractory hypoxemia, respiratory rate and respiratory distress, chest X-ray showed double lung Diffuse infiltration shadow, multiple concurrent multiple organ failure, blood gas analysis showed oxygen index (referring to arterial carbon dioxide partial pressure / forced oxygen absorption) equal to or less than 2617 kPa.

Acute left heart failure caused by severe pulmonary congestion and edema
There are heart-based diseases, symptoms of left heart failure, signs, chest X-ray showed congestion and edema, and sometimes pneumonia, chronic obstructive pulmonary disease (COPD) is difficult to identify, but left ventricular failure in patients with brain sodium Increased peptide levels, and then the two brain natriuretic peptide level is not high, help identify.

Diffuse pulmonary fibrosis
Clinical manifestations of progressive dyspnea accompanied by irritating dry cough, diffuse reticular bacterial shadow in both X-ray and mid-lower lung, pulmonary function tests for restrictive pulmonary ventilation dysfunction.

Pleural disease
Including pleural effusion, pneumothorax, pleural thickening. Outstanding clinical manifestations of these diseases are unrelated to the position of dyspnea, physical examination showed the trachea can be displaced, the chest parts of the intercostal space can be widened or narrowed, percussion was voiced or drum sound, pulmonary function tests for restrictive lung ventilation Dysfunction, chest radiography, chest CT examination of pleural effusion, pneumatosis or pleural thickening performance.

Acute pulmonary embolism
Is due to endogenous or exogenous emboli plug the pulmonary artery or its branches cause clinical symptoms of pulmonary circulatory disorders. There are two basic clinical syndromes: pulmonary infarction: sudden breathing problems, chest pain, hemoptysis, pleural friction sounds, unexplained dyspnea. The disease has caused the formation of emboli or hypercoagulable state of the underlying diseases, chest X-ray showed oval or triangle infiltration of bacteria, severe pulmonary artery prominent, laboratory examination showed increased levels of D-dimer and muscle Calprotectin level is not high, the ECG may be abnormal, heart, pulmonary angiography and radionuclide lung perfusion scan can accuray understand the location and scope of embolism.

Circulatory ischemic disease common in a variety of causes of severe shock, due to decreased peripheral vascular contraction of cardiac output, lack of circulating blood volume, or peripheral artery narrowing, inadequate perfusion of peripheral tissue, hypoxia to the skin mucosa was Blue and purple. Severe neonatal diarrhea, if not treated properly, due to dehydration, can also cause cyanosis. In addition, Raynaud's disease, acromegaly also belong to this category. Raynaud's disease is characterized by numbness or convulsions in both hands or feet, typical pale skin-bruising-flushing Renault at each finger (toe) at onset. Hand, foot and cyanosis is characterized by persistent pre-capillary arteriolar spasm at room temperature, leading to reduced blood flow, skin bruising and skin temperature decreased, infrared thermal imaging screen is dark black or no shadow.

Circulatory congestion around the disease
Such as right heart failure, constrictive pericarditis and local venous lesions (thrombophlebitis, superior vena cava syndrome, varicose veins), its mechanism of cyanosis occurs systemic congestion, peripheral blood flow is slow, oxygen in the surrounding tissue Was taken too much.